Discussions
1
The mechanics of breathing are greatly compromised in each of the cases in the following manners: infant respiratory distress syndrome leads air sacs to collapse when air is expired and increases the energy needed to breathe; emphysema leads to air sacs becoming damaged and enlarged, which makes it difficult to breathe; and pulmonary fibrosis leads to air sacs becoming scarred, which makes it difficult to breathe.
The physiology that plays a vital role in the development of these diseases consists of the following: infant respiratory distress syndrome stems from a lack of surfactant in the lungs; premature birth is often one reason for this lack.[footnoteRef:2] The physiology of the development of emphysema can include the presence of chronic bronchitis which can lead to it; smoking, or exposure to harmful chemicals and irritants over time. For pulmonary fibrosis, the physiology includes exposure to silica dust, asbestos, coal dust, and long term exposure to toxins. [2: Jasani, Bonny, Nandkishor Kabra, and Ruchi Nanavati. "Surfactant replacement therapy beyond respiratory distress syndrome in neonates." Indian pediatrics 53, no. 3 (2016): 229-234.]
The factors that are most important for them to know as they treat patients are to understand the patient’s quality of life and to include the patient in the care process. Quality of care is meaningless if it does not include a conversation about quality of life when it...
Bibliography
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Jasani, Bonny, Nandkishor Kabra, and Ruchi Nanavati. “Surfactant replacement therapy beyond respiratory distress syndrome in neonates.” Indian Pediatrics 53, no. 3 (2016): 229-234.
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Pathophysiology of Gastric Acid Stimulation and Production The human body produces gastric acid in the stomach, primarily to digest proteins (Anand, 2015; Huether & McCance, 2012). It is composed o hydrochloric acid and sodium chloride. It digests proteins through the action of digestive enzymes and allowing digestive enzymes break down the long chains of amino acids from digested proteins. The production of gastric acid us regulated by the autonomous nervous system
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